Chronic Kidney Disease (CKD) is an abnormal kidney function and/or structure that lasts longer than 3 months and is harmful to the health of the affected person. CKD is more precisely defined by several criteria. Changes are manifested by pathological findings in serum and urine, with frequent morphological changes in the kidney and reduction in glomerular filtration rate (GFR). Arterial hypertension (AH) and diabetic nephropathy (DN) are the leading causes of CKD. AH is defined as systolic pressure greater than or equal to 140 mm / Hg and diastolic pressure greater than or equal to 90 mm / Hg. Prevalence of AH in CKD patients is very high and increases with renal impairment. AH and CKD are interconnected in a causal and consequent relationship. Arterial pressure (AP) usually increases with the decline in GFR, while on the other hand, the permanently elevated AT accelerates the development and progression of CKD. Therefore it is very important to treat AH to prevent the onset or progression of CKD. Non-pharmacological treatment of AH in CKD include diet with reduced salt intake, weight reduction in obese people, regular physical activity and decreased alcohol intake. Achieving optimal AP in patients with CKD is often a very demanding task. Medications such as ACE inhibitors, ARBs, diuretics, calcium channel blockers are necessary to postpone and/or stop the progression of CKD and prevent the development of cardiovascular complications.